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Topics - ―λlτεrηιτγ-

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1
Video / Dr. Richard Schulze: The Natural Healing Food Program
« on: July 16, 2021, 10:50:21 pm »
Dr. Richard Schulze: The Natural Healing Food Program

https://youtu.be/6NdlxjspRu4

2
Video / Dr. Richard Schulze - All About The Bowel
« on: July 16, 2021, 08:41:20 pm »
Dr. Richard Schulze - All About The Bowel

https://youtu.be/Lg8L0mw1CJE

3
Video / Dr. Richard Schulze - Natural Healing Crusade
« on: July 16, 2021, 08:37:08 pm »
Dr. Richard Schulze Natural Healing Crusade

Probably the most underrated herbalist I would recommend.

https://youtu.be/8LLEUtLmD3U Part 1

https://youtu.be/lBFCVxR1oCQ Part 2

https://youtu.be/jfkXjAepOlU Part 3

https://youtu.be/p6VpFfYotlI Part 4

https://youtu.be/EJxZNC-UBrQ Part 5

https://youtu.be/ZCwFLBwGgk0 Part 6

https://youtu.be/vSsaXwZrDas Part 7

https://youtu.be/XEHpyDEb5tY Part 8

4
"The scent of magnolia and jasmine is in part due to a compound called methyl dihydrojasmonate. This chemical is produced by the flowers and easily becomes airborne. A group of cell physiologists has now shown that the lining of the human nose has a specific type of receptor called VN1R1. When I stand under the magnolia tree and inhale, airborne methyl dihydrojasmonate molecules are actually binding to the receptors in my nose, which then send signals to my brain. The RUB researchers have collaborated with a team at the University Hospital Dresden to track exactly where those signals are going. Activating the VN1R1 receptor stimulates the areas of the brain controlling the limbic system, which is associated with human emotions, motivation, memory, and smell. It also activates the hypothalamus, especially in women, which controls sexual behaviour by regulating hormone levels. The effect seems to be specific to magnolias and jasmine though. Researchers tested compounds responsible for other floral scents and found they had no effect. It seems magnolias are in fact producing the scent of romance – or at least lust."

"If this is actually the case, and humans produce this compound, it could represent the first example of a human pheromone. Pheromones occur in the vast majority of animals and are a form of scent-based communication whereby one animal produces a compound, and when the other smells it, the compound elicits a specific, consistent physiological or behavioral response. Humans have never been shown to produce pheromones, and proving their existence is an uphill battle. The classic example of pheromones are as sex signals, but human attraction is a complicated affair that is heavily influenced by social conditioning and learned behaviors. The effects of the compound may not be obvious, or replicable across all people, calling into doubt its status as a pheromone. Nevertheless, any compound that stimulates the VN1R1 receptor is a good candidate for the first human pheromone"

https://biophilesblog.wordpress.com/2015/05/19/sensual-scents-how-magnolias-turn-on-the-human-brain/

6
Video / (Herbalist) Chuck Riffinberg | Awfully Irish Podcast
« on: April 15, 2021, 08:14:44 pm »
(Herbalist) Chuck Riffinberg | Awfully Irish Podcast #164
Jan 7, 2021

https://youtu.be/aKIQs7wMyGY

9
Experimental Research / Red Light Therapy
« on: December 12, 2020, 07:18:31 am »
Red light therapy treats pain relief, wound healing, anxiety/depression, hair loss, weight loss, wound healing, fibromyalga, muscle pain, fatigue, skincare, and acne.
It is anti-aging and increases cognitive performance,
They sell cheap led light strips with a remote that goes around the top of your walls.
They can be used for green light and red light therapy.

10
General Discussion / Nitrogen & Oxygen
« on: December 12, 2020, 07:01:43 am »
Quote
How It Works
Your body needs the nitrogen in amino acids from protein foods to make other amino acids it uses to synthesize human proteins, according to Virtual Chembook at Elmhurst College. Not only do your various tissues contain protein, your metabolic processes depend upon enzymes, all of which consist of various kinds of proteins. The nucleic acid DNA, which makes up your genes, and RNA, which is involved in protein synthesis, also contain nitrogen.

Features
Normal growth, cell replacement and tissue repair all require nitrogen for production of new cells. Although nitrogen is abundant in the environment, humans cannot directly use it from the air or soil, but instead depend on microbes and green plants to convert it into form our bodies can use. Your body is constantly recycling nitrogen from amino acids. If amino acids are not used for protein synthesis, they can be broken into components, including nitrogen, to produce energy. Nitrogen can also be used to make other types of compounds that aren't proteins, such as the heme in hemoglobin, which carries oxygen in red blood cells.

Requirements
A healthy adult male needs about 105 milligrams of nitrogen per kilogram, or per 2.2 pounds per day. About 0.83 gram of protein per kilogram per day is considered sufficient to cover nitrogen requirements, according to the International Dairy Foundation. That means a 220-pound man would need 83 grams, or about 3 ounces, of protein a day to supply his nitrogen needs. The breakdown of protein results in ammonia, a nitrogen-containing byproduct that your body eliminates.

Additional Information
Your body eliminates ammonia by converting it to urea, which your kidneys then excrete in urine. In this way, nitrogen is returned to the environment. Unlike populations in underdeveloped countries, Americans do not commonly suffer from a lack of dietary nitrogen unless they are on extreme diets that do not contain sufficient protein. Symptoms of deficiency include hair loss, delayed wound healing, muscle weakness and wasting, brittle hair and hair loss.

Nitrogen in molecules help them to cross the blood-brain barrier,
so I feel like the reason Nitrous Oxide (2 nitrogen atoms attached to an oxygen atom) is so euphoric is cause oxygen is a painkiller and
Quote
"Pain is a lack of oxygen at the cellular level." While I'm not sure this is always true, I believe that in most cases pain is caused by a lack of oxygen reaching the tissues. Usually pain is caused by inflammation, which is the normal response to tissue damage.
Quote
Oxygen should be viewed as a drug. Remember that the difference between therapeutic and toxic is the dose. Chest pain is not an indication for oxygen administration.

I feel like increased oxygen in the air would kill pain and be anti-depressant and anxiolytic.
Since humans started on earth we lost most of the world's plants.
They are what convert our carbon dioxide to oxygen,
so all people should grow plants.
Like pets, they can bring enjoyment.

11
General Discussion / News
« on: December 11, 2020, 09:28:18 pm »
I'm sorry that I have been away.
I could not log into the site;
And I just got my laptop back from the shop, which was out of commission since around the end of May / beginning of June?
I still need another admin to fix my password for me so I can create new topics and such.
I will be continuing to update the site and make posts.
As well as deleting all the bullshit posts from new users with a 1 or 2 post count, that are nonsensical posts to advertise links in their signatures.
Posts of this kind will be removed.

I hope everyone continues visiting back.
If you enjoy reading this website, thank you for the support.
If you can contribute to the forum and start conversations, I encourage you to sign up and post.

Kind regards

15
Supplements / Lithium
« on: January 19, 2020, 06:27:46 pm »
Oxidative metabolism
Quote
Evidence suggests that mitochondrial dysfunction is present in patients with bipolar disorder.[72] Oxidative stress and reduced levels of anti-oxidants (such as glutathione) lead to cell death. Lithium may protect against oxidative stress by up-regulating complex I and II of the mitochondrial electron transport chain.

Dopamine and G-protein coupling
Quote
During mania, there is an increase in neurotransmission of dopamine that causes a secondary homeostatic down-regulation, resulting in decreased neurotransmission of dopamine, which can cause depression.[72] Additionally, the post-synaptic actions of dopamine are mediated through G-protein coupled receptors. Once dopamine is coupled to the G-protein receptors, it stimulates other secondary messenger systems that modulate neurotransmission. Studies found that in autopsies (which do not necessarily reflect living people), people with bipolar disorder had increased G-protein coupling compared to people without bipolar disorder.[72] Lithium treatment alters the function of certain subunits of the dopamine associated G-protein, which may be part of its mechanism of action.

Glutamate and NMDA receptors
Quote
Glutamate levels are observed to be elevated during mania. Lithium is thought to provide long-term mood stabilization and have anti-manic properties by modulating glutamate levels.[72] It is proposed that lithium competes with magnesium for binding to NMDA glutamate receptor, increasing the availability of glutamate in post-synaptic neurons.[72] The NMDA receptor is also affected by other neurotransmitters such as serotonin and dopamine. Effects observed appear exclusive to lithium and have not been observed by other monovalent ions such as rubidium and caesium.

GABA receptors
Quote
GABA is an inhibitory neurotransmitter that plays an important role in regulating dopamine and glutamate neurotransmission.[72] It was found that patients with bipolar disorder had lower GABA levels, which results in excitotoxicity and can cause apoptosis (cell loss). Lithium has been shown to increase the level of GABA in plasma and cerebral spinal fluid.[74] Lithium counteracts these degrading processes by decreasing pro-apoptotic proteins and stimulating release of neuroprotective proteins.[72] Lithium's regulation of both excitatory dopaminergic and glutamatergic systems through GABA may play a role in its mood stabilizing effects.

Cyclic AMP secondary messengers
Quote
Lithium's therapeutic effects are thought to be partially attributable to its interactions with several signal transduction mechanisms.[76][77] The cyclic AMP secondary messenger system is shown to be modulated by lithium. Lithium was found to increase the basal levels of cyclic AMP but impair receptor coupled stimulation of cyclic AMP production.[72] It is hypothesized that the dual effects of lithium are due to the inhibition of G-proteins that mediate cyclic AMP production.[72] Over a long period of lithium treatment, cyclic AMP and adenylate cyclase levels are further changed by gene transcription factors.

Inositol depletion hypothesis
Quote
Lithium treatment has been found to inhibit the enzyme inositol monophosphatase, involved in degrading inositol monophosphate to inositol required in PIP2 synthesis. This leads to lower levels of inositol triphosphate, created by decomposition of PIP2.[78] This effect has been suggested to be further enhanced with an inositol triphosphate reuptake inhibitor. Inositol disruptions have been linked to memory impairment and depression. It is known with good certainty that signals from the receptors coupled to the phosphoinositide signal transduction is affected by lithium.[79] myo-inositol is also regulated by the high affinity sodium mI transport system (SMIT). Lithium is hypothesized to inhibit mI entering the cells and mitigating the function of SMIT.[72] Reductions of cellular levels of myo-inositol results in the inhibition of the phosphoinositide cycle.

https://en.wikipedia.org/wiki/Lithium_(medication)#Glutamate_and_NMDA_receptors

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